1Assistant Department of Forensic Medicine and Pathological Anatomy, Andijan State Medical Institute, Uzbekistan
2Professor, Doctor of Medical Sciences, Director of the Republican Center for Pathological Anatomy, Uzbekistan
3Associate Professor, Candidate of Medical Sciences, Head of the Department of Pediatric Surgery, Andijan State Medical Institute, Uzbekistan
4Doctor of Medical Sciences, Head of The Department of Children, Adolescents and Food Hygiene, Tashkent Medical Academy, Uzbekistan
Online published on 1 May, 2021.
This article presents the pathomorphological changes that develop in the lungs in viral-bacterial pneumonia. The results showed that in viral-bacterial pneumonia, the lungs initially develop hemorrhagic exudative inflammation, which is accompanied by changes characteristic of bacterial pneumonia, purulent-necrotic, serous-fibrinous exudative inflammation, which is diffuse in both lungs; is manifested by the development of polymorphic apparent inflammation involving bronchoalveolar tissue. When streptococcal or staphylococcal lesions are added to the viral lesion, purulent-necrotic processes are added to the hemorrhagic exudative inflammation; foci of inflammation often involve the lower and posterior portions of the lung. The addition of conditionally pathogenic bacteria and fungi indicates the presence of immunodeficiency and other co-morbidities in children, and pneumonia often persists with mixed-type inflammation.
Pathomorphological, Bronchoalveolar, Alveocytes, Aveola, Distirculation, Symplasts, Microvasculitis, Influenza, Parainfluenza, Bronchopneumonia, E.coli, Proteus, Enterobacter, Serracia
